High uric acid—called hyperuricemia—often doesn’t cause pain until it triggers gout or kidney stones. That makes it easy to ignore. This page gives clear, useful steps you can use right away: how to recognize it, what raises uric acid, and realistic things you can do to bring levels down.
Uric acid comes from breaking down purines (parts of many foods and your own cells). Levels rise when your body either makes too much or can’t get rid of enough. Common reasons include:
Blood tests measure serum uric acid. Levels above about 6.8 mg/dL are where crystals can form; many doctors treat people with gout to below 6 mg/dL, and below 5 mg/dL if attacks are frequent.
Small, steady changes work better than extreme diets. Try these steps that most people can follow:
If lifestyle changes aren’t enough, doctors can prescribe medicines that lower uric acid—common ones are allopurinol and febuxostat, which reduce production, or probenecid, which helps the kidneys remove more uric acid. Severe, resistant cases may need stronger options under a specialist’s care.
See a doctor if you have sudden, very painful joint swelling (classic gout), frequent attacks, or kidney stone symptoms. Ask for a serum uric acid test and a clear treatment plan with target levels and follow-up checks. Managing hyperuricemia is usually a mix of steady lifestyle habits and the right medication when needed.
As a gout sufferer, I've been researching different ways to manage the condition, and I came across Allopurinol. This medication is quite effective in reducing uric acid levels, which is the main cause of gout and hyperuricemia. It works by inhibiting an enzyme called xanthine oxidase, responsible for the production of uric acid in our bodies. By taking Allopurinol, I've noticed a significant improvement in my gout symptoms, and my flare-ups have become less frequent. I believe this medicine is a game-changer for people like me who struggle with gout and high uric acid levels.